Isoniazid activity is terminated by bacterial persistence [2]

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dc.contributor.authorMitchison D.A.
dc.contributor.authorJindani A.
dc.contributor.authorDavies G.R.
dc.contributor.authorSirgel F.
dc.date.accessioned2011-05-15T16:03:33Z
dc.date.available2011-05-15T16:03:33Z
dc.date.issued2007
dc.description.abstract[No abstract available]
dc.description.versionLetter
dc.identifier.citationJournal of Infectious Diseases
dc.identifier.citation195
dc.identifier.citation12
dc.identifier.issn221899
dc.identifier.other10.1086/518046
dc.identifier.urihttp://hdl.handle.net/10019.1/12675
dc.subjectisoniazid
dc.subjectantibacterial activity
dc.subjectantibiotic resistance
dc.subjectbacterial growth
dc.subjectbactericidal activity
dc.subjectbacterium culture
dc.subjectclinical feature
dc.subjecthuman
dc.subjectin vitro study
dc.subjectletter
dc.subjectmonotherapy
dc.subjectpersistent infection
dc.subjectpharmacodynamics
dc.subjectphenotype
dc.subjectpriority journal
dc.subjectAntitubercular Agents
dc.subjectDrug Resistance, Bacterial
dc.subjectHumans
dc.subjectIsoniazid
dc.subjectModels, Biological
dc.subjectMycobacterium tuberculosis
dc.subjectTuberculosis
dc.titleIsoniazid activity is terminated by bacterial persistence [2]
dc.typeLetter
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