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Metabolic bone disease in preterm infants

dc.contributor.authorBeyers, Nuldaen_ZA
dc.contributor.authorHough, F. S.en_ZA
dc.date.accessioned2011-03-18T14:57:31Z
dc.date.available2011-03-18T14:57:31Z
dc.date.issued1986
dc.identifier.issn2078-5135 (online)
dc.identifier.issn0256-9574 (print)
dc.identifier.urihttp://hdl.handle.net/10019.1/7469
dc.descriptionCITATION: Beyers, N. & Hough, F. S. 1986. Metabolic bone disease in preterm infants. South African Medical Journal, 70:407-413.
dc.descriptionThe original publication is available at http://www.samj.org.za
dc.description.abstractSmall preterm infants often develop osteopenia with or without rickets and with or without fractures. Whether these bone abnormalities all form part of the same disease process with a wide spectrum of presentation or whether each abnormality represents a different disease is as yet unclear. Bone mineralization depends largely on adequate supplies of calcium and phosphate. The normal intra-uterine accretion of these minerals is higher than can be achieved by feeding preterm babies postnatally with breastmilk or conventional formulas. Supplementation with calcium, phosphorus and vitamin D is needed to prevent the development of 'neonatal osteopathy'. The main action of vitamin D in the preterm baby is probably to increase the intestinal absorption of calcium and phosphorus, although it may, together with other calciotrophic hormones, have a more specific effect on bone growth.
dc.format.extent7 pages
dc.language.isoen
dc.publisherHealth & Medical Publishing Group
dc.subjectBones -- Metabolism -- Disordersen_ZA
dc.subjectPremature infantsen_ZA
dc.titleMetabolic bone disease in preterm infantsen_ZA
dc.typeArticle
dc.description.versionPublisher’s version
dc.rights.holderSouth African Medical Journal


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