p38 and JNK have distinct regulatory functions on the development of apoptosis during simulated ischaemia and reperfusion in neonatal cardiomyocytes.
dc.contributor.author | Engelbrecht A-M | |
dc.contributor.author | Niesler CU | |
dc.contributor.author | Page C | |
dc.contributor.author | Lochner A | |
dc.date.accessioned | 2013-01-23T14:11:12Z | |
dc.date.available | 2013-01-23T14:11:12Z | |
dc.date.issued | 2004 | |
dc.description | Gesondheidswetenskappe | |
dc.description | Geneeskundige Fisiologie | |
dc.description | Please help us populate SUNScholar with the post print version of this article. It can be e-mailed to: scholar@sun.ac.za | |
dc.identifier.citation | Basic Research in Cardiology | |
dc.identifier.uri | http://hdl.handle.net/10019.1/78668 | |
dc.publisher | DR DIETRICH STEINKOPFF VERLAG, PO BOX 10 04 62, DARMSTADT, GERMANY, D-64204 | |
dc.title | p38 and JNK have distinct regulatory functions on the development of apoptosis during simulated ischaemia and reperfusion in neonatal cardiomyocytes. | |
dc.type | Journal Articles (subsidised) |